Cortical parvalbumin GABAergic deficits with α7 nicotinic acetylcholine receptor deletion: implications for schizophrenia.
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Overview
publication date
- January 1, 2014
published in
has subject area
- Age Factors
- Animals
- Animals, Newborn
- Cells, Cultured
- Cerebral Cortex
- Embryo, Mammalian
- Female
- Gene Expression Regulation, Developmental
- Glutamate Decarboxylase
- Inhibitory Postsynaptic Potentials
- Male
- Membrane Potentials
- Mice
- Mice, Inbred C57BL
- Mice, Transgenic
- Neurons
- Parvalbumins
- Receptors, GABA-A
- alpha7 Nicotinic Acetylcholine Receptor
- gamma-Aminobutyric Acid