authors
- Lu, Qiulun
- Ma, Zejun
- Ding, Ye
- Bedarida, Tatiana
- Chen, Liming
- Xie, Zhonglin
- Ping Song, PhD
- Zou, Ming-Hui
Circulating miR-103a-3p contributes to angiotensin II-induced renal inflammation and fibrosis via a SNRK/NF-κB/p65 regulatory axis.
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Overview
publication date
- January 1, 2019
published in
- Nature communications Journal
has subject area
- Adult
- Angiotensin II
- Animals
- Case-Control Studies
- Cells, Cultured
- Disease Models, Animal
- Female
- Fibrosis
- Glomerulonephritis
- Healthy Volunteers
- Humans
- Hypertension, Renal
- Kidney Glomerulus
- Male
- Mice
- Mice, Inbred C57BL
- Mice, Knockout
- MicroRNAs
- Middle Aged
- Nephritis
- Primary Cell Culture
- Protein Serine-Threonine Kinases
- Transcription Factor RelA