authors
- Cai, Weijing
- Schnaider-Beeri, Michal
- Woodward, Mark
- Striker, Gary E
- Vlassara, Helen
- Uribarri, Jaime
- Zhu, Li
- Chen, Xue
- Swamy, Shobha
- Zhao, Zhengshan
- Grosjean, Fabrizio
- Simonaro, Calogera
- George A. Kuchel, MD, FRCP, AGSF
Oral glycotoxins are a modifiable cause of dementia and the metabolic syndrome in mice and humans.
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Overview
publication date
- January 1, 2014
published in
has subject area
- ADAM Proteins
- ADAM10 Protein
- Administration, Oral
- Aged
- Amyloid Precursor Protein Secretases
- Amyloid beta-Peptides
- Animals
- Brain
- Cognition
- Cytokines
- Dementia
- Female
- Gliosis
- Glycation End Products, Advanced
- Humans
- Insulin
- Male
- Membrane Proteins
- Memory
- Metabolic Syndrome
- Mice
- Mice, Inbred C57BL
- Middle Aged
- Nicotinamide Phosphoribosyltransferase
- Oxidative Stress
- PPAR gamma
- Pyruvaldehyde
- Receptor for Advanced Glycation End Products
- Receptors, Immunologic
- Sirtuin 1
- Time Factors
- Transcription, Genetic