authors
- Kandhaya-Pillai, Renuka
- Kirkland, James L
- Oshima, Junko
- Hou, Deyin
- Zhang, Jiaming
- Yang, Xiaomeng
- Compoginis, Goli
- Mori, Takayasu
- Tchkonia, Tamara
- Martin, George M
- Hisama, Fuki M
SMAD4 mutations and cross-talk between TGF-β/IFNγ signaling accelerate rates of DNA damage and cellular senescence, resulting in a segmental progeroid syndrome-the Myhre syndrome.
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